𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Persistence of a component of DNA methylation in gastric mucosae afterHelicobacter pylorieradication

✍ Scribed by Takeshi Nakajima; Shotaro Enomoto; Satoshi Yamashita; Takayuki Ando; Yukihiro Nakanishi; Kazuyuki Nakazawa; Ichiro Oda; Takuji Gotoda; Toshikazu Ushijima


Book ID
106180568
Publisher
Springer Japan
Year
2009
Tongue
English
Weight
370 KB
Volume
45
Category
Article
ISSN
0944-1174

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


The presence of a methylation fingerprin
✍ Takeshi Nakajima; Satoshi Yamashita; Takao Maekita; Tohru Niwa; Kazuyuki Nakazaw πŸ“‚ Article πŸ“… 2009 πŸ› John Wiley and Sons 🌐 French βš– 400 KB πŸ‘ 1 views

## Abstract Aberrant DNA methylation is deeply involved in human cancers, but its inducers and targets are still mostly unclear. __Helicobacter pylori__ infection was recently shown to induce aberrant methylation in gastric mucosae, and produce a predisposed field for cancerization. Here, we analyz

DNA methylation of microRNA genes in gas
✍ Takayuki Ando; Takeichi Yoshida; Shotaro Enomoto; Kiyoshi Asada; Masae Tatematsu πŸ“‚ Article πŸ“… 2009 πŸ› John Wiley and Sons 🌐 French βš– 540 KB

## Abstract Accumulation of aberrant DNA methylation in normal‐appearing gastric mucosae, mostly induced by __H. pylori__ infection, is now known to be deeply involved in predisposition to gastric cancers (epigenetic field defect), and silencing of protein‐coding genes has been analyzed so far. In