✦ LIBER ✦

P4.56 Alleviation of a cacostatic stress response slows disease progression in a mouse model of familial ALS

✍ Scribed by J.C. Dodge; B.K. Kaspar; L.S. Shihabuddin; S.H. Cheng

Book ID: 116794834
Publisher: Elsevier Science
Year: 2011
Tongue: English
Weight: 46 KB
Volume: 21
Category: Article
ISSN: 0960-8966
DOI: 10.1016/j.nmd.2011.06.1021

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Minocycline Slows Disease Progression in

Minocycline Slows Disease Progression in a Mouse Model of Amyotrophic Lateral Sclerosis

✍ Jasna Kriz; Minh Dang Nguyen; Jean-Pierre Julien 📂 Article 📅 2002 🏛 Elsevier Science 🌐 English ⚖ 435 KB

Inhibition of caspase-1 slows disease pr

Inhibition of caspase-1 slows disease progression in a mouse model of Huntington's disease

✍ Friedlander, Robert M.; Ona, Victor O.; Li, Mingwei; Vonsattel, Jean Paul G.; An 📂 Article 📅 1999 🏛 Nature Publishing Group 🌐 English ⚖ 378 KB

Hdac6 deletion delays disease progressio

Hdac6 deletion delays disease progression in the SOD1G93A mouse model of ALS

✍ Taes, I.; Timmers, M.; Hersmus, N.; Bento-Abreu, A.; Van Den Bosch, L.; Van Damm 📂 Article 📅 2013 🏛 Oxford University Press 🌐 English ⚖ 229 KB

The crucial role of caspase-9 in the dis

The crucial role of caspase-9 in the disease progression of a transgenic ALS mouse model

✍ Inoue, H. 📂 Article 📅 2003 🏛 Nature Publishing Group 🌐 English ⚖ 373 KB

Ammonium tetrathiomolybdate delays onset

Ammonium tetrathiomolybdate delays onset, prolongs survival, and slows progression of disease in a mouse model for amyotrophic lateral sclerosis

✍ Eiichi Tokuda; Shin-ichi Ono; Kumiko Ishige; Shunsuke Watanabe; Eriko Okawa; Yos 📂 Article 📅 2008 🏛 Elsevier Science 🌐 English ⚖ 672 KB

Relationship of microglial and astrocyti

Relationship of microglial and astrocytic activation to disease onset and progression in a transgenic model of familial ALS

✍ Edward D. Hall; Jo A. Oostveen; Mark E. Gurney 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 344 KB

Transgenic mice that highly over-express a mutated human CuZn superoxide dismutase (SOD1) gene [gly93-->ala; TgN(SOD1-G93A)G1H line] found in some patients with familial ALS (FALS) have been shown to develop motor neuron disease that is characterized by motor neuron loss in the lumbar and cervical s