The recent development of a rat model of amyotrophic lateral sclerosis (ALS) in which the rats harbor a mutated human SOD1 (G93A) gene has greatly expanded the range of potential experiments, because the rats' large size permits biochemical analyses and therapeutic trials, such as the intrathecal in
โฆ LIBER โฆ
Hdac6 deletion delays disease progression in the SOD1G93A mouse model of ALS
โ Scribed by Taes, I.; Timmers, M.; Hersmus, N.; Bento-Abreu, A.; Van Den Bosch, L.; Van Damme, P.; Auwerx, J.; Robberecht, W.
- Book ID
- 121717519
- Publisher
- Oxford University Press
- Year
- 2013
- Tongue
- English
- Weight
- 229 KB
- Volume
- 22
- Category
- Article
- ISSN
- 0964-6906
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