## Abstract The potential role of __p16__^__INK4a__^ methylation in breast cancer is controversial whereas there are no data on fibroadenoma. To assess if inactivation of __p16__^__INK4a__^ by promoter hypermethylation occurs in this hyperproliferative benign breast lesion or, on the contrary, it i
β¦ LIBER β¦
p16INK4a promoter methylation and 9p21 allelic loss in colorectal carcinomas: relation with immunohistochemical p16INK4a expression and with tumor budding
β Scribed by Friedrich Prall; Christiane Ostwald; Volker Weirich; Horst Nizze
- Book ID
- 116534141
- Publisher
- Elsevier Science
- Year
- 2006
- Tongue
- English
- Weight
- 351 KB
- Volume
- 37
- Category
- Article
- ISSN
- 1532-8392
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## Abstract Routine pathological examination cannot distinctively predict the clinical course of meningiomas because even histologically benign tumors may recur after gross total resection. Numerous efforts have been made for the evaluation of different immunohistochemical assays in meningioma prog