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Oral tolerance in the treatment of inflammatory autoimmune diseases

✍ Scribed by R. M. Wardrop, III; C. C. Whitacre

Publisher
SP Birkhäuser Verlag Basel
Year
1999
Tongue
English
Weight
308 KB
Volume
48
Category
Article
ISSN
1420-908X

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ral tolerance describes the observation that a state of hyporesponsiveness follows immunization with a previously fed protein. It was first described in 1911 when Wells fed hen egg proteins to guinea-pigs and found them resistant to anaphylaxis when challengedL In 1946, Chase fed guinea-pigs the con

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Oral tolerance is the phenomenon of systemic, antigen specific, immunological hyporesponsiveness that results from oral administration of a protein. The mechanism by which tolerance is generated depends on the amount of antigen administered; low doses favor induction of regulatory T cells while high

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## Abstract Autoimmune diseases are characterized by the loss of tolerance toward self‐antigens and the induction of destructive immune responses leading to tissue damage. Most patients with autoimmune diseases are treated with immunosuppressive drugs that suppress the immune response in a non‐spec

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Nuclear factor kB (NF-kB) is a family of structurally related transcription factors. In mammals, the NF-kB family consists of five members: NF-kB1 (p105/50), NF-kB2 (p100/52), RelA (p65), RelB and c-Rel. They all have a structurally conserved N-terminal 300-amino-acid Rel homology domain (RHD), whic

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The vertebrate immune system has evolved to protect against invasive pathogens, but with this adaptation comes the risk of autoimmunity, the recognition and destruction of self tissues by cells of the immune system. To avoid this problem the system has evolved active mechanisms by which tolerance is