No associations of polymorphisms in ADPRT with hepatitis B virus clearance and hepatocellular carcinoma occurrence in a Korean population
β Scribed by Jin Sol Lee; Jeong-Hyun Kim; Byung-Lae Park; Hyun Sub Cheong; InSong Koh; Jason Yong-ha Kim; Tae Joon Park; Charisse Flerida Pasaje; Joon Seol Bae; Hyo-Suk Lee; Yoon Jun Kim; Hyoung Doo Shin
- Book ID
- 118088383
- Publisher
- John Wiley and Sons
- Year
- 2011
- Tongue
- English
- Weight
- 112 KB
- Volume
- 41
- Category
- Article
- ISSN
- 1386-6346
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## Abstract The __RecA homolog__, __E. coli__ (__S. cerevisiae__) (__RAD51__) may modulate hepatitis B virus (HBV) infection by maintaining genome integrity and mediating homologous DNA repairs. In this study, 16 sequence variations were detected by resequencing all exons, the exonβintron boundary,
Some individuals who are chronically infected with hepatitis B virus (HBV) eventually lose hepatitis B surface antigen (HBsAg). Hepatocellular carcinoma (HCC) has been demonstrated to occur in a few patients after loss of HBsAg. Neither factors associated with loss of HBsAg nor the incidence of HCC