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No associations of polymorphisms in ADPRT with hepatitis B virus clearance and hepatocellular carcinoma occurrence in a Korean population

✍ Scribed by Jin Sol Lee; Jeong-Hyun Kim; Byung-Lae Park; Hyun Sub Cheong; InSong Koh; Jason Yong-ha Kim; Tae Joon Park; Charisse Flerida Pasaje; Joon Seol Bae; Hyo-Suk Lee; Yoon Jun Kim; Hyoung Doo Shin


Book ID
118088383
Publisher
John Wiley and Sons
Year
2011
Tongue
English
Weight
112 KB
Volume
41
Category
Article
ISSN
1386-6346

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Lack of association of RAD51 genetic var
✍ Charisse Flerida A. Pasaje; Jeong-Hyun Kim; Byung-Lae Park; Hyun Sub Cheong; Joo πŸ“‚ Article πŸ“… 2011 πŸ› John Wiley and Sons 🌐 English βš– 203 KB

## Abstract The __RecA homolog__, __E. coli__ (__S. cerevisiae__) (__RAD51__) may modulate hepatitis B virus (HBV) infection by maintaining genome integrity and mediating homologous DNA repairs. In this study, 16 sequence variations were detected by resequencing all exons, the exon–intron boundary,

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Some individuals who are chronically infected with hepatitis B virus (HBV) eventually lose hepatitis B surface antigen (HBsAg). Hepatocellular carcinoma (HCC) has been demonstrated to occur in a few patients after loss of HBsAg. Neither factors associated with loss of HBsAg nor the incidence of HCC