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No association ofTFgene polymorphisms with hepatitis B virus Clearance and hepatocellular carcinoma occurrence in a Korean population

✍ Scribed by Jason Yongha Kim; Jeong-Hyun Kim; Tae Joon Park; Jin Sol Lee; Charisse Flerida Pasaje; Joon Seol Bae; Byung-Lae Park; Hyun Sub Cheong; Hyo-Suk Lee; Yoon Jun Kim; Hyoung Doo Shin


Book ID
107693573
Publisher
The Genetics Society of Korea
Year
2011
Tongue
English
Weight
313 KB
Volume
33
Category
Article
ISSN
1976-9571

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## Abstract The __RecA homolog__, __E. coli__ (__S. cerevisiae__) (__RAD51__) may modulate hepatitis B virus (HBV) infection by maintaining genome integrity and mediating homologous DNA repairs. In this study, 16 sequence variations were detected by resequencing all exons, the exon–intron boundary,

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Some individuals who are chronically infected with hepatitis B virus (HBV) eventually lose hepatitis B surface antigen (HBsAg). Hepatocellular carcinoma (HCC) has been demonstrated to occur in a few patients after loss of HBsAg. Neither factors associated with loss of HBsAg nor the incidence of HCC