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Neuronal and glial accumulation of α- and β-synucleins in human lipidoses

✍ Scribed by Kyoko Suzuki; Eizo Iseki; Takashi Togo; Akira Yamaguchi; Omi Katsuse; Kayoko Katsuyama; Seiichi Kanzaki; Kazumasa Shiozaki; Chiaki Kawanishi; Sumimasa Yamashita; Yukichi Tanaka; Shoji Yamanaka; Yoshio Hirayasu

Book ID
106073564
Publisher
Springer-Verlag
Year
2007
Tongue
English
Weight
619 KB
Volume
114
Category
Article
ISSN
0001-6322

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## Abstract Parkinson's disease is neuropathologically characterized by the presence of Lewy bodies, whose major component is α‐synuclein. We had previously generated transgenic mice that expressed human α‐synuclein carrying an Ala53Thr point mutation (hα‐syn140m) under the control of the rat tyros