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Myocardial protection evoked by hyperoxic exposure involves signaling through nitric oxide and mitogen activated protein kinases

✍ Scribed by A. Ruusalepp; G. Czibik; T. Flatebø; J. Vaage; G. Valen


Book ID
106067013
Publisher
Springer
Year
2007
Tongue
English
Weight
487 KB
Volume
102
Category
Article
ISSN
0300-8428

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## Abstract Low‐level activation of __N__‐methyl‐d‐aspartate receptors (NMDARs) results in a decrease in the ability of tetanic stimulation to induce long‐term potentiation (LTP). This NMDAR‐mediated LTP inhibition is observed with low micromolar concentrations of NMDA or chelation of ambient extra