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MyD88 Is Dispensable for Cerebral Amyloidosis and Neuroinflammation in APP/PS1 Transgenic Mice

โœ Scribed by Weitz, Tara M.; Gate, David; Rezai-Zadeh, Kavon; Town, Terrence


Book ID
126758834
Publisher
American Society for Investigative Pathology
Year
2014
Tongue
English
Weight
1011 KB
Volume
184
Category
Article
ISSN
0002-9440

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## Abstract Amyloid deposits are one of the hallmarks of Alzheimer's disease (AD), one of the most devastating neurodegenerative disorders. In transgenic mice modeling Alzheimer's pathology, the MR transverse relaxation time (__T__~2~) has been described to be modulated by amyloidosis. This modific