✦ LIBER ✦

Moyamoya-induced paroxysmal dyskinesia

✍ Scribed by Pedro Gonzalez-Alegre; Zakaria Ammache; Patricia H. Davis; Robert L. Rodnitzky

Publisher: John Wiley and Sons
Year: 2003
Tongue: English
Weight: 315 KB
Volume: 18
Category: Article
ISSN: 0885-3185
DOI: 10.1002/mds.10483

No coin nor oath required. For personal study only.

✦ Synopsis

Abstract

Moyamoya disease (MMD) is an uncommon intracranial vasculopathy that typically presents with ischemic or hemorrhagic stroke. Persistent choreoathetosis has been identified as a rare early manifestation of MMD. We present 2 patients with paroxysmal dyskinesia as the initial symptom of MMD, one resembling paroxysmal kinesigenic dyskinesia (PKD) and the other paroxysmal non‐kinesigenic dyskinesia (PNKD). We also review the cases of moyamoya‐induced chorea reported previously, none of which resembled PKD or PNKD. We hypothesize that both hormonal and ischemic factors may be implicated in the pathogenesis of these abnormal involuntary movements. These cases suggest that MMD should be included in the differential diagnosis of PKD and PNKD. © 2003 Movement Disorder Society

📜 SIMILAR VOLUMES

Paroxysmal dyskinesias

✍ Kailash P. Bhatia 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 126 KB

## Abstract Paroxysmal movement disorders are a relatively rare and heterogenous group of conditions manifesting as episodic dyskinesia lasting a brief duration. Three forms are clearly recognized, namely, paroxysmal kinesigenic (PKD), nonkinisegenic (PNKD), and exercise induced (PED). There have b

Secondary paroxysmal dyskinesias

✍ Jaishri Blakeley; Joseph Jankovic 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 89 KB

Paroxysmal dyskinesias (PxDs) are involuntary, episodic movements that include paroxysmal kinesigenic (PKD), paroxysmal nonkinesigenic (PNKD), and paroxysmal hypnogenic (PHD) varieties. Although most PxDs are primary (idiopathic or genetic), we found 17 of our 76 patients with PxD (22%) to have an i

Paroxysmal dyskinesias in mice

✍ Thomas L. Shirley; Lekha M. Rao; Ellen J. Hess; H. A. Jinnah 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 139 KB

## Abstract Animal models of human disease are important tools for revealing the underlying mechanisms of pathophysiology and developing therapeutic strategies. Several unique mouse calcium channel mutants have been identified with nonepileptic, episodic dyskinetic movements that are phenotypically

Alcohol-induced paroxysmal nonkinesogeni

Alcohol-induced paroxysmal nonkinesogenic dyskinesia after pallidal hypoxic insult

✍ Graham T.A. Warner; John H. McAuley 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 199 KB

## Abstract We describe the first case of paroxysmal nonkinesogenic dyskinesia secondary to pallidal ischaemia, which is uniquely and specifically triggered by alcohol. © 2002 Movement Disorder Society

Paroxysmal kinesigenic dyskinesia in inf

Paroxysmal kinesigenic dyskinesia in infancy

✍ Steven Frucht; Stanley Fahn 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 312 KB 👁 1 views

GLUT1 gene mutations cause sporadic paro

GLUT1 gene mutations cause sporadic paroxysmal exercise-induced dyskinesias

✍ Susanne A. Schneider; Coro Paisan-Ruiz; Ines Garcia-Gorostiaga; Niall P. Quinn; 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 218 KB

## Abstract Paroxysmal exercise‐induced dyskinesias (PED) are involuntary intermittent movements triggered by prolonged physical exertion. Autosomal dominant inheritance may occur. Recently, mutations in the glucose transporter 1 (__GLUT1__) gene (chr. 1p35–p31.3) have been identified as a cause in