Modulation of growth and apoptosis response in PC-3 and LNCAP prostate-cancer cell lines by FAS
β Scribed by Takumi Takeuchi; Yukihiro Sasaki; Tetsuo Ueki; Takahiro Kaziwara; Nobuo Moriyama; Kazuki Kawabe; Tadao Kakizoe
- Publisher
- John Wiley and Sons
- Year
- 1996
- Tongue
- French
- Weight
- 627 KB
- Volume
- 67
- Category
- Article
- ISSN
- 0020-7136
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Background: Exposure of prostate carcinoma cell lines to retinoids, which function through the classical retinoic acid nuclear receptor, (rars) or retinoid x receptors (rxrs), results in minimal cytostatic inhibition of cell proliferation. ## Methods: Growth inhibition and various regulatory r
## Abstract Genistein is a major component of soybean isoflavone and has multiple functions resulting in antitumor effects. Prostate cancer is 1 of the targets for the preventive role of genistein. We examined the effect of genistein on human prostate cancer (LNCaP and PCβ3) cells. Proliferation of
## Abstract It is widely known that death receptor Fasβdependent apoptotic signals are associated with development of prostate cancer, but the key pathways involved in sensitivity to the apoptosis remain unclear. Here we investigated the molecular mechanism by which 2βmethoxyestradiol (2βME) effect
data indicate that antiprogestins and antiestrogens could inhibit prostate cancer cell growth in vitro and in vivo. The main objective of the present studies was to explore the role of bcl 2 and TGFβ€ 1 for induction of apoptosis in LNCaP prostate cancer cells growing in culture as a treatment respon
BACKGROUND. Prostatic secretory protein of 94 amino acids (PSP94) is one of the predominant proteins found in human seminal fluid. Limited information is available regarding a physiological function for PSP94. An important step in the elucidation of this function is the determination of the mechanis