## Abstract ## Objective Treatment of rheumatoid arthritis (RA) with tumor necrosis factor (TNF)–blocking agents, including etanercept and infliximab, has resulted in reductions in the radiographic progression of RA. However, the exact mechanism by which this protection occurs has not been determi
Modulation of CD28 expression with anti–tumor necrosis factor α therapy in rheumatoid arthritis
✍ Scribed by Ewa Bryl; Abbe N. Vallejo; Eric L. Matteson; Jacek M. Witkowski; Cornelia M. Weyand; Jorg J. Goronzy
- Publisher
- John Wiley and Sons
- Year
- 2005
- Tongue
- English
- Weight
- 218 KB
- Volume
- 52
- Category
- Article
- ISSN
- 0004-3591
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract ## Objective Neovascularization, with an increased number of synovial vessels with a characteristic morphology, seems to contribute to the progression of psoriatic arthritis (PsA). Accordingly, angiogenesis may be an important therapeutic target in PsA. The aim of this study was to ana
COPD ϭ chronic obstructive pulmonary disease. 300 Letters \* RA ϭ rheumatoid arthritis; JRA ϭ juvenile rheumatoid arthritis; ESR ϭ erythrocyte sedimentation rate; CRP ϭ C-reactive protein; VAS ϭ visual analog scale; GH ϭ general health; DAS28 ϭ Disease Activity Score in 28 joints; HAQ ϭ Health Asses
The authors are the principal investigators of the British Society of Rheumatology (BSR) Biologics Register. The BSR receives financial support for the Register from the manufacturers of biologic agents currently licensed for use in the treatment of patients with inflammatory rheumatic diseases. The