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Modulation of acetylcholine receptor expression in seronegative myasthenia gravis

โœ Scribed by Sandrine Poea; Thierry Guyon; Jocelyne Bidault; Corinne Bruand; Vincent Mouly; Sonia Berrih-Aknin

Publisher
John Wiley and Sons
Year
2000
Tongue
English
Weight
545 KB
Volume
48
Category
Article
ISSN
0364-5134

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Acetylcholine receptor expression in hum
Acetylcholine receptor expression in human extraocular muscles and their susceptibility to myasthenia gravis
โœ C. MacLennan; Dr. D. Beeson; A.-M. Buijs; A. Vincent; J. Newsom-Davis ๐Ÿ“‚ Article ๐Ÿ“… 1997 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 906 KB

In myasthenia gravis (MG), extraocular muscle (EOM) weakness is often an initial and persisting symptom. It has been proposed that acetylcholine receptor (AChR) from EOM is antigenically different from AChR of other innervated muscles and that the presence of antibodies to fetal AChR expressed in EO

Modulation of the anti-acetylcholine rec
Modulation of the anti-acetylcholine receptor response and experimental autoimmune myasthenia gravis by recombinant fragments of the acetylcholine receptor
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Myasthenia gravis (MG) is a neuromuscular disorder of man caused by a humoral response to the acetylcholine receptor (AChR). Most of the antibodies in MG and in experimental autoimmune myasthenia gravis (EAMG) are directed to the extracellular portion of the AChR alpha subunit, and within it, primar