Mitf cooperates with Rb1 and activates p21Cip1 expression to regulate cell cycle progression
β Scribed by Carreira, Suzanne; Goodall, Jane; Aksan, Isil; La Rocca, S. Anna; Galibert, Marie-Dominique; Denat, Laurence; Larue, Lionel; Goding, Colin R.
- Book ID
- 109893791
- Publisher
- Nature Publishing Group
- Year
- 2005
- Tongue
- English
- Weight
- 507 KB
- Volume
- 433
- Category
- Article
- ISSN
- 0028-0836
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
## Abstract Ribosomal biogenesis is correlated with cell cycle, cell proliferation, cell growth and tumorigenesis. Some oncogenes and tumor suppressors are involved in regulating the formation of mature ribosome and affecting the ribosomal biogenesis. In previous studies, the mitochondrial ribosoma
Insulin-like growth factor (IGF)-1 is accumulated in the diabetic kidney and is considered to be involved in the development of glomerular sclerosis. Here, we investigate IGF-1 regulation of laminin, an extracellular matrix (ECM) component, and cyclin D1 and p21 Cip1 , cell-cycle progression factor,
## Abstract We have used HeLa cells without mitochondrial DNA (Ο^0^βcells) and transient Ο^0^βphenocopies, obtained from wildβtype cells by shortβterm treatment with ethidium bromide, to analyze how the absence of a functional mitochondrial respiratory chain slows down proliferation. We ruled out a