Pathological processes affecting presynaptic terminals may contribute to morbidity following traumatic brain injury (TBI). Posttraumatic widespread neuronal depolarization and elevated extracellular potassium and glutamate are predicted to alter the transduction of action potentials in terminals int
Long-term potentiation deficits and excitability changes following traumatic brain injury
β Scribed by Thomas M. Reeves; Bruce G. Lyeth; John T. Povlishock
- Publisher
- Springer-Verlag
- Year
- 1995
- Tongue
- English
- Weight
- 875 KB
- Volume
- 106
- Category
- Article
- ISSN
- 0014-4819
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