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Lipid peroxidation and protein modification in a mouse model of chronic iron overload

✍ Scribed by Mark A. Sochaski; Wally J. Bartfay; Suzanne R. Thorpe; John W. Baynes; Emma Bartfay; Denis C. Lehotay; Peter P. Liu

Book ID
117828533
Publisher
Elsevier Science
Year
2002
Tongue
English
Weight
113 KB
Volume
51
Category
Article
ISSN
1532-8600

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To study the effect of iron-overload on hepatic lipid peroxidation, two rat models of haemochromatosis were employed: in the first model resembling secondary haemochromatosis, repeated i.p. injections with Fe-dextran led to an accumulation of Fe in Kupffer cells, while in the second model resembling

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In both hereditary hemochromatosis (HHC) and in the various forms of secondary hemochromatosis, there is a pathological expansion of total body iron stores, due mainly to an inappropriate increase in absorption of dietary iron (1-3). The liver is the major recipient of this excess absorbed iron, and