## Abstract Radiographic mammary microcalcifications are one of the most pertinent diagnostic markers of breast cancer. Breast tissue calcification in the form of calcium hydroxyapatite (HA) is strongly associated with malignant disease. We tested the hypothesis that calcium HA may exert biological
Laminin induces matrix metalloproteinase-9 expression and activation in human cervical cancer cell line (SiHa)
✍ Scribed by Gargi Maity; Triparna Sen; Amitava Chatterjee
- Publisher
- Springer-Verlag
- Year
- 2010
- Tongue
- English
- Weight
- 744 KB
- Volume
- 137
- Category
- Article
- ISSN
- 1432-1335
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
## Abstract Matrilysin, which is a member of the matrix metalloproteinase family and is implicated in colon cancer invasion, is expressed in human colon adenocarcinoma—derived SW1116 cells. We investigated the effect of α‐difluoromethylornithine (DFMO) on matrilysin expression in this cell line bec
## Abstract The plasminogen‐activating (PA) and matrix metalloproteinase (MMP) enzyme systems are implicated in proteolytic turnover of the extracellular matrix (ECM) associated with biologic processes including wound healing, inflammation and angiogenesis. Aberrant expression of components of the
## Abstract The ubiquitin‐proteasome pathway (UPP) is involved in the degradation of the extracellular matrix (ECM) and trophoblastic invasion during early pregnancy. Our previous studies demonstrated that inhibition of UPP suppresses expression of matrix metalloproteinase (MMP)‐2 and ‐9. LMP2 is a
## Abstract Matrix metalloproteinase‐9 (MMP‐9) plays a critical role in tumor invasion and metastasis. Here, we investigate the effect of fibroblast growth factor‐1 (FGF‐1) on the expression of MMP‐9 in ENU1564, an ethyl‐__N__‐nitrosourea‐induced rat mammary adenocarcinoma cell line. We observed th
Numerous reports have shown an association between overexpression of the epidermal growth factor receptor (EGFR), and poor prognosis in head and neck squamous cell carcinomas (HNSCC), however, the underlying mechanisms are still unclear. In the present study, we set out to determine whether EGFR exp