𝔖 Bobbio Scriptorium
✦   LIBER   ✦

L1CAM–integrin interaction induces constitutive NF-κB activation in pancreatic adenocarcinoma cells by enhancing IL-1β expression

✍ Scribed by Kiefel, H; Bondong, S; Erbe-Hoffmann, N; Hazin, J; Riedle, S; Wolf, J; Pfeifer, M; Arlt, A; Schäfer, H; Müerköster, S Sebens

Book ID
109972420
Publisher
Nature Publishing Group
Year
2010
Tongue
English
Weight
868 KB
Volume
29
Category
Article
ISSN
0950-9232

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

IL-1β induces urokinse-plasminogen activ
IL-1β induces urokinse-plasminogen activator expression and cell migration through PKCα, JNK1/2, and NF-κB in A549 cells
✍ Ching-Yi Cheng; Hsi-Lung Hsieh; Chi-Chin Sun; Chih-Chung Lin; Shue-Fen Luo; Chue 📂 Article 📅 2009 🏛 John Wiley and Sons 🌐 English ⚖ 516 KB

## Abstract Breakdown of the extracellular matrix (ECM) is accomplished by the concerted action of several proteases, including the urokinase plasminogen‐activator (uPA) system and matrix metalloproteinases (MMPs), which is crucial for cancer invasion and metastasis. Several reports have shown that

IL-1β, an immediate early protein secret
IL-1β, an immediate early protein secreted by activated microglia, induces iNOS/NO in C6 astrocytoma cells through p38 MAPK and NF-κB pathways
✍ Yun-Jung Kim; So-Young Hwang; Eok-Soo Oh; Seikwan Oh; Inn-Oc Han 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 324 KB

## Abstract In the present study we sought to examine cell–cell interactions by investigating the effects of factors released by stimulated microglia on inducible nitric oxide (NO) synthase (iNOS) induction in astrocytoma cells. After examining the temporal profiles of proinflammatory molecules ind