𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Inactivation of retinoic acid receptor β by promoter CpG hypermethylation in gastric cancer

✍ Scribed by Ken Hayashi; Hiroshi Yokozaki; Naohide Oue; Tetsuo Suzuki; Wataru Yasui; Eiichi Tahara; Steve Goodison; Reuben Lotan

Book ID
114428693
Publisher
John Wiley and Sons
Year
2001
Tongue
English
Weight
208 KB
Volume
68
Category
Article
ISSN
0301-4681

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Inactivation of helicase-like transcript
Inactivation of helicase-like transcription factor by promoter hypermethylation in human gastric cancer
✍ Wai K. Leung; Jun Yu; Alfa H.C. Bai; Michael W.Y. Chan; Ka-Kui Chan; Ka-Fai To; 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 154 KB

## Abstract Helicase‐like transcription factor (__HLTF__), a member of the SWI/SNF (mating type switching/sucrose nonfermenting) chromatin‐remodeling complex, is recently found to be inactivated by promoter hypermethylation in human colorectal cancer. However, the role of this putative tumor suppre

CpG hypermethylation of promoter region
CpG hypermethylation of promoter region and inactivation of E-cadherin gene in human bladder cancer
✍ Leopoldo Alves Ribeiro-Filho; Joseph Franks; Masahiro Sasaki; Hiroaki Shiina; Lo 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 384 KB

## Abstract Several studies have shown that E‐cadherin expression is lost during malignant transformation. We hypothesized that CpG methylation in the promoter region may inactivate the expression of the E‐cadherin gene in human bladder cancer. Normal and bladder cancer samples from 51 patients wer

Epigenetic inactivation of SOCS-1 by CpG
Epigenetic inactivation of SOCS-1 by CpG island hypermethylation in human gastric carcinoma
✍ Yasuhiro Oshimo; Kazuya Kuraoka; Hirofumi Nakayama; Yasuhiko Kitadai; Kazuhiro Y 📂 Article 📅 2004 🏛 John Wiley and Sons 🌐 French ⚖ 460 KB

## Abstract __Suppressor of cytokine signaling (SOCS)‐1__ inhibits signaling of the Janus kinase (JAK)/signal transducers and activators of transcription (STAT) pathway by several cytokines and has tumor suppressor activity. Methylation of the __SOCS‐1__ CpG island has been shown to inactivate the