The purpose of these experiments was to test the hypothesis that impaired glucose-stimulated insulin secretion in NIDDM is due to mutations in the islet beta cell/liver glucose transporter (GLUT 2) gene. Using oligonucleotide primers flanking each of the 11 exons, the structural portion of the gene
โฆ LIBER โฆ
Importance of the GLUT2 glucose transporter for pancreatic beta cell toxicity of alloxan
โ Scribed by R., Munday; S., Lenzen; M., Elsner; M., Tiedge; B., Guldbakke
- Book ID
- 113017614
- Publisher
- Springer
- Year
- 2002
- Tongue
- English
- Weight
- 187 KB
- Volume
- 45
- Category
- Article
- ISSN
- 0012-186X
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