## Abstract Cellular and molecular mechanisms of methamphetamine (METH)βinduced neurotoxicity may involve alterations of cellular redox status and induction of inflammatory genes in endothelial cells. To study these hypotheses, molecular signaling pathways of METHβinduced inflammatory responses via
β¦ LIBER β¦
Hypoxia induces AP-1-regulated genes and AP-1 transcription factor binding in human endothelial and other cell types
β Scribed by Ram S. Bandyopadhyay; Michael Phelan; Douglas V. Faller
- Book ID
- 113263394
- Publisher
- Elsevier Science
- Year
- 1995
- Tongue
- English
- Weight
- 668 KB
- Volume
- 1264
- Category
- Article
- ISSN
- 0167-4781
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