## Abstract Over the past 12 years, a wide variety of neurodegenerative diseases has been linked to mutations in mitochondrial genes located in either the mitochondrial DNA (mtDNA) or the nuclear DNA (nDNA). These disorders encompass an array of unorthodox inheritance patterns and a plethora of sym
β¦ LIBER β¦
Human complex I defects in neurodegenerative diseases
β Scribed by A.H.V Schapira
- Book ID
- 117047323
- Publisher
- Elsevier Science
- Year
- 1998
- Tongue
- English
- Weight
- 96 KB
- Volume
- 1364
- Category
- Article
- ISSN
- 0005-2728
No coin nor oath required. For personal study only.
π SIMILAR VOLUMES
Mitochondrial defects in neurodegenerati
β
Wallace, Douglas C.
π
Article
π
2001
π
John Wiley and Sons
π
English
β 199 KB
Colloquium C017: Axonal Transport Defect
π
Article
π
2006
π
John Wiley and Sons
π
English
β 76 KB
Axonal transport defects: a common theme
β
Subhojit Roy; Bin Zhang; Virginia M.-Y. Lee; John Q. Trojanowski
π
Article
π
2005
π
Springer-Verlag
π
English
β 470 KB
Modeling human neurodegenerative disease
β
Miguel A. Gama Sosa; Rita De Gasperi; Gregory A. Elder
π
Article
π
2011
π
Springer
π
English
β 913 KB
Assembly factors of human mitochondrial
β
Matthew Mckenzie; Michael T. Ryan
π
Article
π
2010
π
John Wiley and Sons
π
English
β 159 KB
## Abstract NADHβubiquinone oxidoreductase (complex I) is a large, multimeric enzyme complex involved in the generation of ATP by oxidative phosphorylation. Complex I is comprised of 45 subunits which must be assembled together in a coordinated process to form the mature holoenzyme. In recent years
Protein misfolding in neurodegenerative
β
E. I. Agorogiannis; G. I. Agorogiannis; A. Papadimitriou; G. M. Hadjigeorgiou
π
Article
π
2004
π
John Wiley and Sons
π
English
β 107 KB