✦ LIBER ✦

Homocarnosine levels in cerebrospinal fluid of patients with infantile spasms under ACTH therapy

✍ Scribed by Ohtsuka, Chikaya; Sato, Yasuko; Takahashi, Hiroshi

Book ID: 122428513
Publisher: Elsevier Science
Year: 1983
Tongue: English
Weight: 436 KB
Volume: 5
Category: Article
ISSN: 0387-7604
DOI: 10.1016/S0387-7604(83)80075-8

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Prolactin levels in cerebrospinal fluid

Prolactin levels in cerebrospinal fluid of patients with infantile spasms

✍ Güzide Burça Aydln; Gülşen Köse; Aydan Değerli̇yurt; Necmettin Di̇n; Deniz Çamur 📂 Article 📅 2002 🏛 Elsevier Science 🌐 English ⚖ 47 KB

Decreased cerebrospinal fluid levels of

Decreased cerebrospinal fluid levels of β-endorphin and ACTH in children with infantile spasms

✍ S. Nagamitsu; T. Matsuishi; Y. Yamashita; T. Shimizu; R. Iwanaga; Y. Murakami; M 📂 Article 📅 2001 🏛 Springer 🌐 English ⚖ 91 KB

Excitatory amino acid levels in cerebros

Excitatory amino acid levels in cerebrospinal fluid of patients with infantile spasms

✍ E. İnce; U. Karagöl; G. Deda 📂 Article 📅 1997 🏛 John Wiley and Sons 🌐 English ⚖ 393 KB

Studies on the Levels of Cortisol in Pla

Studies on the Levels of Cortisol in Plasma and Cerebrospinal Fluid in Patients with Infantile Spasms

✍ Kyoko Hagishima 📂 Article 📅 1972 🏛 John Wiley and Sons 🌐 English ⚖ 117 KB

Kynurenic acid is decreased in cerebrosp

Kynurenic acid is decreased in cerebrospinal fluid of patients with infantile spasms

✍ Yamamoto, Hitoshi; Shindo, Ikuyo; Egawa, Bunsei; Horiguchi, Kumiko 📂 Article 📅 1994 🏛 Elsevier Science 🌐 English ⚖ 314 KB

A patient with infantile spasms and low

A patient with infantile spasms and low homovanillic acid levels in cerebrospinal fluid:l-dopa dependent seizures?

✍ H. Sugie; Y. Sugie; N. Kato; Y. Fukuyama 📂 Article 📅 1989 🏛 Springer 🌐 English ⚖ 221 KB

We report a 3-month-old female with infantile spasms that responded transiently to pyridoxine and permanently to oral L-dopa. Initial CSF levels of homovanillic acid were low, suggesting disturbed turnover of dopamine. These findings suggest that disturbed brain monoamine metabolism may be causally