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High prevalence of GB virus C/hepatitis G virus infection among the Jewish population in Uzbekistan

✍ Scribed by Takanobu Kato; Masashi Mizokami; Tatsunori Nakano; Yutaka Kondo; Ken-ichi Ohba; Etsuro Orito; Ryuzo Ueda; Motokazu Mukaide; Vladimir Gurtsevitch; Alexander Syrtsev; Ruslan Ruzibakiev; Mamur Abdurakhnamov; Masahiro Yamashita; Masanori Hayami


Book ID
117366349
Publisher
Elsevier Science
Year
1997
Tongue
English
Weight
447 KB
Volume
48
Category
Article
ISSN
0168-1702

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Prevalence of GB virus-C/hepatitis G vir
✍ Skidmore, Susan J.; Collingham, Kathryn E. 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 48 KB 👁 2 views

It is difficult to explain the high levels of infection seen with GBV-C/HGV if transmission relies on the parenteral route. A group of young women was investigated in order to establish the prevalence of infection in this age group of the general population and perhaps indicate other possible routes

High prevalence of GB virus C/hepatitis
✍ Carmen L. Loureiro; Roberto Alonso; Belkis A. Pacheco; María G. Uzcátegui; L. Vi 📂 Article 📅 2002 🏛 John Wiley and Sons 🌐 English ⚖ 85 KB

## Abstract GB virus C or hepatitis G virus (GBV‐C/HGV) is highly prevalent among population groups at risk of parenterally transmitted viral agents, but it has also a worldwide distribution in other non‐risk population groups. GBV‐C/HGV RNA and antibodies against its envelope protein (anti‐E2 Abs)

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✍ Gallian, P.; Rodrigues, V.; Cantaloube, J. F.; Dessein, H.; de Micco, P.; Dessei 📂 Article 📅 1998 🏛 John Wiley and Sons 🌐 English ⚖ 139 KB 👁 2 views

A study of GB-C virus/Hepatitis G virus (GBV-C/ HGV) infection was carried out in a rural population of Northeastern Brazil, in which the prevalence of schistosomiasis is 80-90%. Despite the absence of parenteral risk exposure, the prevalence of GBV-C/HGV markers of infection was found to be unusual

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✍ Rey, D.; Fraize, S.; Vidinic, J.; Meyer, P.; Fritsch, S.; Labouret, N.; Schmitt, 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 137 KB 👁 2 views

Prevalence of GBV-C/HGV was determined in a cohort of HIV-infected patients, via a reverse transcription-polymerase chain reaction detection of RNA in serum, amplifying the NS5 region of GBV-C/HGV genome. GBV-C/HGV RNA was detected in 143 (37.7%) of 379 patients, with similar results in the differen