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Hepatic iron overload may contribute to hypertriglyceridemia and hypercholesterolemia in copper-deficient rats

โœ Scribed by Meira Fields; Charles G. Lewis


Book ID
117180246
Publisher
Elsevier Science
Year
1997
Tongue
English
Weight
575 KB
Volume
46
Category
Article
ISSN
1532-8600

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In both hereditary hemochromatosis (HHC) and in the various forms of secondary hemochromatosis, there is a pathological expansion of total body iron stores, due mainly to an inappropriate increase in absorption of dietary iron (1-3). The liver is the major recipient of this excess absorbed iron, and