𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Growth factor responsiveness of human articular chondrocytes in aging and development

✍ Scribed by Pierre-andré Guerne; Francisco Blanco; André Kaelin; Alain Desgeorges; Martin Lotz

Publisher
John Wiley and Sons
Year
1995
Tongue
English
Weight
779 KB
Volume
38
Category
Article
ISSN
0004-3591

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Growth factor responsiveness of human ar
Growth factor responsiveness of human articular chondrocytes: Distinct profiles in primary chondrocytes, subcultured chondrocytes, and fibroblasts
✍ Pierre-André Guerne; Arthur Sublet; Martin Lotz 📂 Article 📅 1994 🏛 John Wiley and Sons 🌐 English ⚖ 991 KB

The objectives of this study were to establish a growth factor response profile for adult human articular chondrocytes, to determine whether this is unique for chondrocytes or influenced by the differentiation status of the cells, and to characterize growth factor interactions. It is shown that tran

Response of articular chondrocytes to pi
Response of articular chondrocytes to pituitary fibroblst growth factor (FGF)
✍ Barton L. Sachs; Victor M. Goldberg; Roland W. Moskowitz; Charles J. Malemud 📂 Article 📅 1982 🏛 John Wiley and Sons 🌐 English ⚖ 997 KB

## Abstract Rabbit chondrocytes from pooled articular joints have been delineated by their time of attachment of culture flasks after initiation of primary monolayer culture, either attached (48‐AT) or floating (48‐F) after 48 hours. A general population of chrondrocytes (attached after 72 hours, 7

Differential expression of TGFβ isoforms
Differential expression of TGFβ isoforms by human articular chondrocytes in response to growth factors
✍ Peter M. Villiger; Martin Lotz 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 883 KB

Transforming growth factor P (TGFP) is a family of important regulators of chondrocyte growth and differentiation. Although TGFP has been detected in cartilage, the TGFj3 isoform5 expressed by chondrocytes and their regulation by growth factors are unknown. This study shows that human articular chon

Interleukin-4 antagonizes oncostatin M a
Interleukin-4 antagonizes oncostatin M and transforming growth factor beta-induced responses in articular chondrocytes
✍ Mohammed El Mabrouk; Hamid Yaqoob Qureshi; Wen Qing Li; Judith Sylvester; Muhamm 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 381 KB

## Abstract Oncostatin M (OSM) stimulates cartilage degradation in rheumatoid arthritis (RA) by inducing matrix metalloproteinases (MMPs) and aggrecanases (ADAMTS; a disintegrin and metalloproteinase with thrombospondin motif). Transforming growth factor beta (TGF‐β1) induces cartilage repair in jo