The purpose of these experiments was to test the hypothesis that impaired glucose-stimulated insulin secretion in NIDDM is due to mutations in the islet beta cell/liver glucose transporter (GLUT 2) gene. Using oligonucleotide primers flanking each of the 11 exons, the structural portion of the gene
✦ LIBER ✦
Glucose transporter 2 gene polymorphisms and beta-cell function in isolated human pancreatic islets
✍ Scribed by S. Del Guerra; R. Lupi; V. D’Aleo; F. Filipponi; U. Boggi; P. Marchetti
- Book ID
- 116404819
- Publisher
- Masson Editeur
- Year
- 2009
- Tongue
- French
- Weight
- 140 KB
- Volume
- 35
- Category
- Article
- ISSN
- 1262-3636
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