Ginsenosides compound K and Rh2 inhibit tumor necrosis factor-α-induced activation of the NF-κB and JNK pathways in human astroglial cells
✍ Scribed by Kyungsun Choi; Myungsun Kim; Jeonghee Ryu; Chulhee Choi
- Book ID
- 116769655
- Publisher
- Elsevier Science
- Year
- 2007
- Tongue
- English
- Weight
- 645 KB
- Volume
- 421
- Category
- Article
- ISSN
- 0304-3940
No coin nor oath required. For personal study only.
📜 SIMILAR VOLUMES
In response to the bacterial endotoxin, LPS, Kupffer cells are induced to express NO and TNF-␣. These compounds are involved in hepatic inflammation/injury, especially that associated with endotoxic shock. In this study, we demonstrate that ebselen (2-phenyl-1,2-benzisoselenazol-3[2H]one), a selenoo
Background: High levels of proinflammatory cytokines are linked to pathogenesis of diarrhea in inflammatory bowel disease (IBD). Na þ absorption is compromised in IBD. The studies were designed to determine the effect of tumor necrosis factor-a (TNFa) on the expression and activity of NHE2, a Na þ /