𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Genetic proof for the transient nature of the Th17 phenotype

✍ Scribed by Florian C. Kurschus; Andrew L. Croxford; André P. Heinen; Simone Wörtge; Daniele Ielo; Ari Waisman


Publisher
John Wiley and Sons
Year
2010
Tongue
English
Weight
470 KB
Volume
40
Category
Article
ISSN
0014-2980

No coin nor oath required. For personal study only.


📜 SIMILAR VOLUMES


Evidence of the transient nature of the
✍ Lorenzo Cosmi; Rolando Cimaz; Laura Maggi; Veronica Santarlasci; Manuela Capone; 📂 Article 📅 2011 🏛 John Wiley and Sons 🌐 English ⚖ 535 KB

## Abstract ## Objective To investigate the phenotype and function of CD4+ T cells in synovial fluid (SF) from the affected joints of children with oligoarticular‐onset juvenile idiopathic arthritis (JIA), and to establish a possible link with disease activity. ## Methods CD4+ T cells were obtai

Quantifying the contribution of genetic
✍ Martina Müller; Angela Döring; Helmut Küchenhoff; Claudia Lamina; Dörthe Malzahn 📂 Article 📅 2008 🏛 John Wiley and Sons 🌐 English ⚖ 188 KB

## Abstract Particularly in studies based on population representative samples, it is of major interest what impact a genetic variant has on the phenotype of interest, which cannot be answered by mere association estimates alone. One possible measure for quantifying the phenotype's variance explain

The nature of the genetic determinant fo
✍ Nugent, M. E. ;Hedges, R. W. 📂 Article 📅 1979 🏛 Springer 🌐 English ⚖ 438 KB

In two unrelated plasmids of incompat-Table 1 ibility groups FII and N the gene for the SHV-1 fl-lactamase exists as part of a transposable element Plasmid of molecular weight 9.5 megadaltons. This transposon has moved onto plasmids of at least three incompatibility groups; PI, Ia and J. This confir

On the nature of genetic changes require
✍ Pieter Uys; Paul D. van Helden 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 112 KB

## Abstract It is clear that genetic mutations are necessary for the development of cancer, but the exact number required is not clear, with estimates ranging from one critical hit (e.g., __p53__) to dozens or perhaps even hundreds of expression changes (by microarray analysis) or chromosomal aberr