๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Genetic analysis of von Willebrand's disease in two large pedigrees: A multivariate approach

โœ Scribed by Goldin, Lynn R. ;Elston, Robert C. ;Graham, John B. ;Miller, Connie H. ;Murphy, Edmond A.

Publisher
John Wiley and Sons
Year
1980
Tongue
English
Weight
844 KB
Volume
6
Category
Article
ISSN
0148-7299

No coin nor oath required. For personal study only.

๐Ÿ“œ SIMILAR VOLUMES

Linkage of familial alzheimer disease to
Linkage of familial alzheimer disease to chromosome 14 in two large early-onset pedigrees: Effects of marker allele frequencies on lod scores
โœ Nechiporuk, A. ;Fain, P. ;Kort, E. ;Nee, L. E. ;Frommelt, E. ;Polinsky, R. J. ;K ๐Ÿ“‚ Article ๐Ÿ“… 1993 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 405 KB ๐Ÿ‘ 2 views

Alzheimer disease (AD) is a devastating neurodegenerative disease leading to global dementia. In addition to sporadic forms of AD, familial forms (FAD) have been recognized. Mutations in the amyloid precursor protein (APP) gene on chromosome (CHR) 21 have been shown to cause early-onset AD in a smal

An approach to the multivariate analysis
An approach to the multivariate analysis of high-density-lipoprotein cholesterol in a large kindred: The Bogalusa Heart Study
โœ C. I. Amos; A. F. Wilson; P. A. Rosenbaum; S. R. Srinivasan; L. S. Webber; R. C. ๐Ÿ“‚ Article ๐Ÿ“… 1986 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 901 KB

The genetic determination of highdensity-lipoprotein cholesterol (HDL-C) levels was evaluated using segregation analysis techniques in a large multigenerational kindred with a high prevalence of coronary heart disease and myocardial infarction. Univariate segregation analysis of HDL-C levels with th

Confronting complexity in late-onset Alz
Confronting complexity in late-onset Alzheimer disease: application of two-stage analysis approach addressing heterogeneity and epistasis
โœ Tricia A. Thornton-Wells; Jason H. Moore; Eden R. Martin; Margaret A. Pericak-Va ๐Ÿ“‚ Article ๐Ÿ“… 2008 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 298 KB

## Abstract Common diseases with a genetic basis are likely to have a very complex etiology, in which the mapping between genotype and phenotype is far from straightforward. A new comprehensive statistical and computational strategy for identifying the missing link between genotype and phenotype ha