𝔖 Bobbio Scriptorium
✦   LIBER   ✦

FunctionalIFNGpolymorphism in intron 1 in association with an increased risk to promote sporadic breast cancer

✍ Scribed by Anjana Saha; Ashish Dhir; Anand Ranjan; Vibhuti Gupta; Narendra Bairwa; Ramesh Bamezai

Publisher
Springer-Verlag
Year
2005
Tongue
English
Weight
160 KB
Volume
57
Category
Article
ISSN
0093-7711

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Functional variants in the promoter of i
Functional variants in the promoter of interleukin-1Ξ² are associated with an increased risk of breast cancer: A case-control analysis in a Chinese population
✍ Jiyong Liu; Xiangjun Zhai; Guangfu Jin; Zhibin Hu; Shui Wang; Xuechen Wang; Jian πŸ“‚ Article πŸ“… 2006 πŸ› John Wiley and Sons 🌐 French βš– 91 KB πŸ‘ 1 views

## Abstract Interleukin 1Ξ² (IL‐1Ξ²) is a multifunctional cytokine that upregulates the inflammatory response, and participates in carcinogenesis, malignant transformation, tumor growth, invasion and metastasis. Two potentially functional polymorphisms (T‐31C and C‐511T) in the __IL‐1__Ξ² gene promote

Variant genotypes of CDKN1A and CDKN1B a
Variant genotypes of CDKN1A and CDKN1B are associated with an increased risk of breast cancer in Chinese women
✍ Hongxia Ma; Guangfu Jin; Zhibin Hu; Xiangjun Zhai; Wensen Chen; Shui Wang; Xuech πŸ“‚ Article πŸ“… 2006 πŸ› John Wiley and Sons 🌐 French βš– 105 KB πŸ‘ 2 views

## Abstract p21^Cip1^ and p27^Kip1^ are cyclin‐dependent kinase inhibitors, which can arrest cell proliferation and serve as tumor suppressors. Reduced protein expression of p21^Cip1^ and p27^Kip1^ was frequently observed in a subset of cancers, including breast cancer. In this study, we hypothesiz

MSI in endometrial carcinoma: Absence of
MSI in endometrial carcinoma: Absence of MLH1 promoter methylation is associated with increased familial risk for cancers
✍ Alison J. Whelan; Sheri Babb; David G. Mutch; Janet Rader; Thomas J. Herzog; Chr πŸ“‚ Article πŸ“… 2002 πŸ› John Wiley and Sons 🌐 French βš– 123 KB πŸ‘ 2 views

## Abstract Loss of DNA mismatch repair occurs in a variety of malignancies and is associated with genome‐wide instability of microsatellite repeats, a molecular phenotype referred to as microsatellite instability (MSI). MSI is a consistent feature of colorectal and endometrial tumors from patients