𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Fructose-1,6-bisphosphate preserves intracellular glutathione and protects cortical neurons against oxidative stress

✍ Scribed by Zinaida S. Vexler; Arthur Wong; Carla Francisco; Catherine Manabat; Stephan Christen; Martin Täuber; Donna M. Ferriero; George Gregory

Book ID
113998831
Publisher
Elsevier Science
Year
2003
Tongue
English
Weight
544 KB
Volume
960
Category
Article
ISSN
0006-8993

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

γ-glutamylcysteine ethyl ester-induced u
γ-glutamylcysteine ethyl ester-induced up-regulation of glutathione protects neurons against Aβ(1–42)-mediated oxidative stress and neurotoxicity: Implications for Alzheimer's disease
✍ Debra Boyd-Kimball; Rukhsana Sultana; Hafiz Mohmmad Abdul; D. Allan Butterfield 📂 Article 📅 2005 🏛 John Wiley and Sons 🌐 English ⚖ 249 KB 👁 1 views

## Abstract Glutathione (GSH) is an important endogenous antioxidant found in millimolar concentrations in the brain. GSH levels have been shown to decrease with aging. Alzheimer's disease (AD) is a neurodegenerative disorder associated with aging and oxidative stress. Aβ(1–42) has been shown to in

Acetyl-L-carnitine-induced up-regulation
Acetyl-L-carnitine-induced up-regulation of heat shock proteins protects cortical neurons against amyloid-beta peptide 1–42-mediated oxidative stress and neurotoxicity: Implications for Alzheimer's disease
✍ Hafiz Mohmmad Abdul; Vittorio Calabrese; Menotti Calvani; D. Allan Butterfield 📂 Article 📅 2006 🏛 John Wiley and Sons 🌐 English ⚖ 636 KB

Alzheimer's disease (AD) is a progressive neurodegenerative disorder characterized by loss of memory and cognition and by senile plaques and neurofibrillary tangles in brain. Amyloid-beta peptide, particularly the 42-amino-acid peptide (Abeta(1-42)), is a principal component of senile plaques and is