𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Fasting-induced intestinal damage is mediated by oxidative and inflammatory responses

✍ Scribed by S. Abdeen; T. C. Mathew; I. Khan; H. Dashti; S. Asfar

Publisher
John Wiley and Sons
Year
2009
Tongue
English
Weight
466 KB
Volume
96
Category
Article
ISSN
0007-1323

No coin nor oath required. For personal study only.

πŸ“œ SIMILAR VOLUMES

Transient inflammatory response induced
Transient inflammatory response induced by apoptotic cells is an important mediator of melanoma cell engraftment and growth
✍ Mariangela Correa; Joel Machado Jr.; CΓ©lia R.W. Carneiro; JoΓ£o Bosco Pesquero; M πŸ“‚ Article πŸ“… 2005 πŸ› John Wiley and Sons 🌐 French βš– 553 KB

## Abstract Two murine melanoma cell lines, Tm1 and Tm5, were derived from a nontumorigenic lineage of pigmented murine melanocytes, melan‐a. Both Tm1 and Tm5 are invariably tumorigenic in syngeneic mice when inoculated s.c. in doses higher than 10^4^ cells; 10^3^ or fewer cells rarely give rise to

Light-dependent mutagenesis by benzo[a]p
Light-dependent mutagenesis by benzo[a]pyrene is mediated via oxidative DNA damage
✍ Su-Ryang Kim; Kiyoko Kokubo; Keiko Matsui; Noriyo Yamada; Yusuke Kanke; Masamich πŸ“‚ Article πŸ“… 2005 πŸ› John Wiley and Sons 🌐 English βš– 168 KB

## Abstract Benzo[__a__]pyrene (B[__a__]P) is an environmental carcinogenic polycyclic aromatic hydrocarbon (PAH). Mammalian enzymes such as cytochrome P‐450s and epoxide hydrase convert B[__a__]P to reactive metabolites that can covalently bind to DNA. However, some carcinogenic compounds that nor

Oxidative damage to sarcoplasmic reticul
Oxidative damage to sarcoplasmic reticulum Ca2+-pump induced by Fe2+/H2O2/ascorbate is not mediated by lipid peroxidation or thiol oxidation and leads to protein fragmentation
✍ Roger F. Castilho; Paulo C. Carvalho-Alves; Anibal E. Vercesi; SΓ©rgio T. Ferreir πŸ“‚ Article πŸ“… 1996 πŸ› Springer 🌐 English βš– 904 KB

The major protein in the sarcoplasmic reticulum (SR) membrane is the Ca2+ transporting ATPase which carries out active Ca2+ pumping at the expense of ATP hydrolysis. The aim of this work was to elucidate the mechanisms by which oxidative stress induced by Fenton's reaction (Fe(2+)+H2O2-->HO.+OH-+Fe3