Excessive nitric oxide (NO) generated by hepatic cells in response to lipopolysaccharide (LPS) and inflammatory substances (e.g., platelet-activating factor [PAF]) is a key contributor to the pathophysiological outcomes observed in the liver during sepsis. In rats subjected to liver-focused endotoxe
Facilitation or suppression of fusimotor activity induced by changes in temperature of the midbrain reticular formation of rats
β Scribed by Haruhiko Sato; Takeshi Hashitani
- Publisher
- Elsevier Science
- Year
- 1989
- Tongue
- English
- Weight
- 987 KB
- Volume
- 14
- Category
- Article
- ISSN
- 0306-4565
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## Abstract A single oral dose of diβ__n__βbutyl phthalate (DBP) to male rats caused a sloughing of the germ cells at 6 h, with more severe sloughing at 24 and 48 h. DBP is metabolized to monoβ__n__βbutyl phthalate (MBP), which is transported through the bloodβtubular barrier into the seminiferous
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