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Expression of p16INK4a in relation to histopathology and viral load of ‘high-risk’ HPV types in cervical neoplastic lesions

✍ Scribed by Sonia Andersson; Darawalee Wangsa; Carmen Flores-Staino; Hamzah Safari; Miriam Mints; Anders Hjerpe; Bjorn Hagmar; Bo Johansson

Book ID
116429101
Publisher
Elsevier Science
Year
2006
Tongue
English
Weight
271 KB
Volume
42
Category
Article
ISSN
0959-8049

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The fragile histidine triad (FHIT) gene encompasses the common chromosomal fragile site FRA3B. Human papilloma virus (HPV), which is the main aetiological agent in cervical cancers, has been found to be able to integrate its genes into the chromosome 3 fragile site of cultured cells, deleting a piec