Gap junctional intercellular communication (GJIC) has been proposed as a cellular mechanism for tumour suppression and there is experimental evidence in support of this. If aberrant GJIC contributes to the formation of human breast tumours, one might expect that the connexins (gap junction proteins)
Expression of connexin 43 and connexin 32 gap-junction proteins in epilepsy-associated brain tumors and in the perilesional epileptic cortex
β Scribed by Eleonora Aronica; Jan A. Gorter; Gerard H. Jansen; Sieger Leenstra; Bulent Yankaya; Dirk Troost
- Publisher
- Springer-Verlag
- Year
- 2001
- Tongue
- English
- Weight
- 381 KB
- Volume
- 101
- Category
- Article
- ISSN
- 0001-6322
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