✦ LIBER ✦

Experimental cerebral hypoperfusion induces white matter injury and microglial activation in the rat brain

✍ Scribed by Eszter Farkas; Gergely Donka; RobA.I. Vos; András Mihály; Ferenc Bari; PaulG.M. Luiten

Publisher: Springer-Verlag
Year: 2004
Tongue: English
Weight: 628 KB
Volume: 108
Category: Article
ISSN: 0001-6322
DOI: 10.1007/s00401-004-0864-9

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Chronic cerebral hypoperfusion induces w

Chronic cerebral hypoperfusion induces white matter lesions and loss of oligodendroglia with DNA fragmentation in the rat

✍ H. Tomimoto; M. Ihara; H. Wakita; R. Ohtani; J.-X. Lin; I. Akiguchi; M. Kinoshit 📂 Article 📅 2003 🏛 Springer-Verlag 🌐 English ⚖ 604 KB

α-Phenyl-n-tert-butyl-nitrone reduces li

α-Phenyl-n-tert-butyl-nitrone reduces lipopolysaccharide-induced white matter injury in the neonatal rat brain

✍ Lir-Wan Fan; Helen J. Mitchell; Lu-Tai Tien; Baoying Zheng; Yi Pang; Philip G. R 📂 Article 📅 2008 🏛 Wiley (John Wiley & Sons) 🌐 English ⚖ 549 KB

ATPase activities in myelin and oligoden

ATPase activities in myelin and oligodendrocytes isolated from the brains of developing rats and from bovine brain white matter

✍ Thomas R. Zimmerman Jr.; Dr. Wendy Cammer 📂 Article 📅 1982 🏛 John Wiley and Sons 🌐 English ⚖ 599 KB

## Abstract Of the Na, K‐ATPase activity in brain homogenates from 20‐, 60‐, and 120‐day‐old rats, 1.4 to 2.6% was recovered in myelin. The relative specific activities, at 0.2 to 0.3 times the specific activities in the rat brain homogenates, did not decrease during development, and myelin from bo

Nerve growth factor and choline acetyltr

Nerve growth factor and choline acetyltransferase activity levels in the rat brain following experimental impairment of cerebral glucose and energy metabolism

✍ Dr. R. Hellweg; R. Nitsch; C. Hock; M. Jaksch; S. Hoyer 📂 Article 📅 1992 🏛 John Wiley and Sons 🌐 English ⚖ 806 KB

C.H.), Federal Republic of Germany Intracerebroventricular (ICV) injection of streptozotocin (STZ) has been reported to impair cerebral glucose utilization and energy metabolism (Nitsch and Hoyer: Neurosci Lett, 128:199-202,1991) and also to prejudice passive avoidance learning in adult rats (Mayer