𝔖 Bobbio Scriptorium
✦   LIBER   ✦

ERK Pathway Mediates the Activation of Cdk2 in IGF-1–Induced Proliferation of Human Osteosarcoma MG-63 Cells

✍ Scribed by Wenyu Zhang; John C. Lee; Sanjay Kumar; Maxine Gowen

Book ID
111486519
Publisher
American Society for Bone and Mineral Research
Year
1999
Tongue
English
Weight
238 KB
Volume
14
Category
Article
ISSN
0884-0431

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Effect of ciprofloxacin on the prolifera
Effect of ciprofloxacin on the proliferation of osteoblast-like MG-63 human osteosarcoma cells in vitro
✍ Theodore Miclau; Matthew L. Edin; Gayle E. Lester; Ronald W. Lindsey; Laurence E 📂 Article 📅 1998 🏛 Elsevier Science 🌐 English ⚖ 390 KB 👁 1 views

## Abstract Locally applied antibiotic therapy is gaining popularity for the treatment of infections associated with open fractures and posttraumatic osteomyelitis. With use of local techniques, ciprofloxacin levels as high as 1,300 μg/ml. or over 200 times the bone levels achieved with intravenous

Mediation of glucocorticoid receptor fun
Mediation of glucocorticoid receptor function by the activation of latent transforming growth factor beta 1 in MG-63 human osteosarcoma cells
✍ Jocelyn Boulanger; Carlos Reyes-Moreno; Michael Koutsilieris 📂 Article 📅 1995 🏛 John Wiley and Sons 🌐 French ⚖ 912 KB

## Abstract We analyzed glucocortcoid receptor function using ligand binding assays, DNA band‐shift analysis and __trans__‐activation of the murine mammary tumor virus‐thymidine kinase‐chloramphenicol acetyltransferase reporter gene in transiently transfected MG‐63 human osteosarcoma cells. Dexamet

IGF-I plus E2 induces proliferation via
IGF-I plus E2 induces proliferation via activation of ROS-dependent ERKs and JNKs in human breast carcinoma cells
✍ Cheng-Wei Lin; Liang-Yo Yang; Shing-Chuan Shen; Yen-Chou Chen 📂 Article 📅 2007 🏛 John Wiley and Sons 🌐 English ⚖ 507 KB

## Abstract Induction of 17β‐estradiol (E2) and insulin‐like growth factor‐I (IGF‐I) has been detected in breast carcinoma, however the interaction between E2 and IGF‐I in the proliferation of breast carcinoma cells is still unclear. In the present study, we found that IGF‐I enhances the E2‐induced