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Enhanced GABA release in cerebral cortical slices derived from rats with thioacetamide-induced hepatic encephalopathy

✍ Scribed by Urszula Wysmyk; Simo S. Oja; Pirjo Saransaari; Jan Albrecht

Publisher: Springer
Year: 1992
Tongue: English
Weight: 316 KB
Volume: 17
Category: Article
ISSN: 0364-3190
DOI: 10.1007/bf00968397

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## Abstract Increased ammonia is a major pathogenic factor in hepatic encephalopathy (HE), a neurologic syndrome associated with glutamatergic dysfunction. Previous studies have shown that in rat cerebral cortical slices or a glia‐derived cell line, acute treatment with ammonia in vitro and in vivo