𝔖 Bobbio Scriptorium
✦   LIBER   ✦

Effects of DA agonist in Huntington disease hyperkinesia

✍ Scribed by T. A. Caraceni; F. Girotti; P. Giovannini; M. Pederzoli; E. A. Parati


Publisher
Springer Milan
Year
1979
Tongue
English
Weight
485 KB
Volume
1
Category
Article
ISSN
1590-1874

No coin nor oath required. For personal study only.


πŸ“œ SIMILAR VOLUMES


Erythrocyte membrane alterations in Hunt
✍ Butterfield, D. A. ;Braden, M. L. ;Markesbery, W. R. πŸ“‚ Article πŸ“… 1978 πŸ› Wiley (John Wiley & Sons) 🌐 English βš– 373 KB

## Abstract The interaction of the inhibitory neurotransmitter γ‐aminobutyric acid (GABA) with erythrocyte membranes from patients with Huntington disease and normal controls has been studied by electron spin resonance. GABA affects the physical state of erythrocyte membrane proteins in control and

Effect of external cueing on gait in Hun
✍ Arnaud Delval; Pierre Krystkowiak; Marie Delliaux; Jean-Louis Blatt; Philippe De πŸ“‚ Article πŸ“… 2008 πŸ› John Wiley and Sons 🌐 English βš– 190 KB πŸ‘ 1 views

## Abstract In Huntington's disease (HD) patients, gait is characterized by a timing disorder with marked intraindividual variability in temporal gait parameters (caused by the presence of both hyperkinetic and hypokinetic features). We sought to determine the influence of use of a metronome on gai

Genetic analysis of Huntington disease i
✍ Ajmar, F. ;Mandich, P. ;Bellone, E. ;Abbruzzese, G. πŸ“‚ Article πŸ“… 1991 πŸ› John Wiley and Sons 🌐 English βš– 374 KB πŸ‘ 1 views

## Abstract Twelve Italian families with Huntington disease were tested with 10 probes known to be linked to the disease locus and able to detect polymorphisms at the following loci on chromosome 4: D4S10, D4S127, D4S95, D4S43, D4S115, D4S111, D4S90. The results confirmed the applicability of the l

Effects of serotonin 5-HT1A agonist in a
✍ William Bara-Jimenez; Francesco Bibbiani; Michael J. Morris; Tzvetelina Dimitrov πŸ“‚ Article πŸ“… 2005 πŸ› John Wiley and Sons 🌐 English βš– 69 KB

## Abstract Intermittent stimulation of striatal dopaminergic receptors seems to contribute to motor dysfunction in advanced Parkinson's disease (PD). With severe dopaminergic denervation, exogenous levodopa is largely decarboxylated to dopamine in serotonergic terminals. If 5‐HT1A autoreceptors re