๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Effects of cardiotrophin-1 (CT-1) in a mouse motor neuron disease

โœ Scribed by Hiroshi Mitsumoto; Bogdan Klinkosz; Erik P. Pioro; Kazufumi Tsuzaka; Takeo Ishiyama; Rhona M. O'Leary; Diane Pennica


Publisher
John Wiley and Sons
Year
2001
Tongue
English
Weight
391 KB
Volume
24
Category
Article
ISSN
0148-639X

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Histometric effects of ciliary neurotrop
โœ Ken Ikeda; Vivien Wong; Thomas H. Holmlund; Tom Greene; Jesse M. Cedarbaum; Rona ๐Ÿ“‚ Article ๐Ÿ“… 1995 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 858 KB

We investigated the histological effects of ciliary neurotrophic factor on degenerating motor neurons, their axons, and skeletal muscles in 68 wobbler mice with motor neuron disease. Treatment consisted of recombinant rat or human ciliary neurotrophic factor (or a vehicle solution), 1-mg/kg subcutan

Protective effect of a novel free radica
โœ K. Abe; S. Morita; T. Kikuchi; Y. Itoyama ๐Ÿ“‚ Article ๐Ÿ“… 1997 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 159 KB

A possible protective effect of a novel free radical scavenger, OPC-14117, on the progressive motor neuron death in wobbler mice was examined. Clinical parameters such as mortality, body weight, motor activity as a rolling number of circular cage, and forelimb muscle power as grasping on a rolling c

Motor neuron disease in a patient with a
โœ Dr E. Nobile-Orazio; G. Legname; R. Daverio; M. Carpo; A. Giuliani; S. Sonnino; ๐Ÿ“‚ Article ๐Ÿ“… 1990 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 505 KB

I n a patient with motor neuron disease and benign IgMk monoclonal gammopathy, the M protein reacted with the glycolipids GM1, GDlb, and asialo GM1 and, by immunoblot, with some high-molecular-weight neural-specific glycoproteins. T h e main reactive bands had an approximate molecular weight of 250

Loss of synaptophysin-positive boutons o
โœ Da Wei Zang; Elizabeth C. Lopes; Surindar S. Cheema ๐Ÿ“‚ Article ๐Ÿ“… 2005 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 255 KB

## Abstract Amyotrophic lateral sclerosis (ALS) is a common form of motor neuron disease (MND) that involves both upper and lower nervous systems. In the SOD1^G93A G1H^ transgenic mouse, a widely used animal model of human ALS, a significant pathology is linked to the degeneration of lower motor ne