## Abstract ## Objective The enhanced expression of experimental arthritis in the absence of interferon‐γ (IFNγ) suggests that IFNγ suppresses arthritis. Interleukin‐17 (IL‐17) is a pivotal T cell cytokine in arthritis, and in vitro studies have indicated that IFNγ suppresses IL‐17 production. We
Effector mechanisms of interleukin-17 in collagen-induced arthritis in the absence of interferon-γ and counteraction by interferon-γ
✍ Scribed by Hilde Kelchtermans, Evelien Schurgers, Lies Geboes…
- Book ID
- 120706906
- Publisher
- BioMed Central
- Year
- 2009
- Tongue
- English
- Weight
- 771 KB
- Volume
- 11
- Category
- Article
- ISSN
- 1478-6354
- DOI
- 10.1186/ar2787
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