✦ LIBER ✦

Disorders of neutrophil function in children with recurrent pyogenic infections

✍ Scribed by F. Patrone; F. Dallegri; E. Bonvini; F. Minervini; C. Sacchetti

Publisher: Springer-Verlag
Year: 1982
Tongue: English
Weight: 483 KB
Volume: 171
Category: Article
ISSN: 0300-8584
DOI: 10.1007/bf02124918

No coin nor oath required. For personal study only.

📜 SIMILAR VOLUMES

Neutrophil bactericidal function is defe

Neutrophil bactericidal function is defective in patients with recurrent urinary tract infections

✍ Condron, C. ;Casey, R. G. ;Shaffii, M. ;Creagh, T. ;Bouchier-Hayes, D ;Toomey, D 📂 Article 📅 2003 🏛 Springer 🌐 English ⚖ 291 KB

Transient abnormal neutrophil functions

Transient abnormal neutrophil functions in a patient with recurrent infection

✍ S. Traniello; R. Carletti; S. Spisani 📂 Article 📅 1981 🏛 Springer-Verlag 🌐 English ⚖ 568 KB

Recurrent pyogenic infections in individ

Recurrent pyogenic infections in individuals with absence of the second component of complement

✍ Hugh A. Sampson; Andreas M. Walchner; Patricia J. Baker 📂 Article 📅 1982 🏛 Springer 🌐 English ⚖ 815 KB

The effects of colchicine on neutrophil

The effects of colchicine on neutrophil function in subjects with recurrent aphthous stomatitis

✍ S Altinor; S Öztürkcan; MM Hah 📂 Article 📅 2003 🏛 John Wiley and Sons 🌐 English ⚖ 568 KB

Efficacy of a polyvalent bacterial lysat

Efficacy of a polyvalent bacterial lysate in children with recurrent respiratory tract infections

✍ Samuel Bentes Ruah; Carlos Ruah; Antje van Aubel; Stephan Abel; Ulrich Elsasser 📂 Article 📅 2001 🏛 Springer Healthcare Communications 🌐 English ⚖ 118 KB

Alveolar macrophages of children sufferi

Alveolar macrophages of children suffering from recurrent infections of respiratory tract are less efficient in eliminating apoptotic neutrophils

✍ J. Pryjma; Jolanta Kaszuba-Zwoinska; J. Pawlik; A. Pogorzelski; G. Lis; J. Baran 📂 Article 📅 1999 🏛 John Wiley and Sons 🌐 English ⚖ 470 KB 👁 1 views

During bacterial infections of the respiratory tract, neutrophils (PMN) are recruited to the lung by various mechanisms, including production of interleukin-8 (IL-8) by alveolar macrophages (AM). After fulfilling their defense function, PMN become apoptotic and have to be disposed of by AM to preven