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Differential expression and signaling of CBL and CBL-B in BCR/ABL transformed cells

✍ Scribed by Sattler, Martin; Pride, Yuri B; Quinnan, Laura R; Verma, Shalini; Malouf, Nicole A; Husson, Hervé; Salgia, Ravi; Lipkowitz, Stanley; Griffin, James D

Book ID: 110066412
Publisher: Nature Publishing Group
Year: 2002
Tongue: English
Weight: 314 KB
Volume: 21
Category: Article
ISSN: 0950-9232
DOI: 10.1038/sj.onc.1205202

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## Abstract Chronic myeloid leukemia (CML) arises as a consequence of the expression of a chimeric fusion protein, p210BCR‐ABL1, which is localized to the cytoplasm and has constitutive protein tyrosine kinase activity. Extensive publications report that p210BCR‐ABL1 complexed with multiple cytopla