## Abstract Many tumors express CD95L (CD178, FasL, APOβ1L) and may thus kill tumorβinfiltrating lymphocytes, a phenomenon called tumor counterattack. However, presently it is not clear whether tumor counterattack is a relevant immune escape mechanism. To characterize the effect of CD95L expression
Dichloroacetate induces tumor-specific radiosensitivity in vitro but attenuates radiation-induced tumor growth delay in vivo
β Scribed by Zwicker, F.; Kirsner, A.; Peschke, P.; Roeder, F.; Debus, J.; Huber, P.E.; Weber, K.J.
- Book ID
- 120590053
- Publisher
- Springer
- Year
- 2013
- Tongue
- German
- Weight
- 893 KB
- Volume
- 189
- Category
- Article
- ISSN
- 0179-7158
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