## Abstract ## BACKGROUND. Tobacco smoking, alcohol abuse, and high‐risk human papillomavirus (HPV) are risk factors in the etiology of oropharyngeal squamous cell carcinomas (SCCs). The __TP53__ polymorphism, in which an arginine (R) is changed to proline (P) at codon 72, is functionally signific
✦ LIBER ✦
Detection of the human papillomavirus and analysis of the TP53 polymorphism of exon 4 at codon 72 in penile squamous cell carcinomas
✍ Scribed by O Humbey; S Cairey-Remonnay; J.S Guérrini; M.P Algros; C Mougin; H Bittard; F Aubin
- Book ID
- 117659266
- Publisher
- Elsevier Science
- Year
- 2003
- Tongue
- English
- Weight
- 165 KB
- Volume
- 39
- Category
- Article
- ISSN
- 0959-8049
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## Abstract The polymorphisms of the tumor suppressor gene p53 have been extensively investigated in numerous malignant tumors, particularly carcinomas associated with human papillomavirus (HPV) infection. However, the results remain controversial. To address a potential correlation between the p53