๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Depolarization or glutamate receptor activation blocks apoptotic cell death of cultured cerebellar granule neurons

โœ Scribed by Guang-Mei Yan; Binhui Ni; Michael Weller; Katherine A. Wood; Steven M. Paul


Book ID
113127199
Publisher
Elsevier Science
Year
1994
Tongue
English
Weight
778 KB
Volume
656
Category
Article
ISSN
0006-8993

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Role of oxidative stress in the apoptoti
โœ Antonio Valencia; Julio Morรกn ๐Ÿ“‚ Article ๐Ÿ“… 2001 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 411 KB ๐Ÿ‘ 2 views

When cultured cerebellar granule neurons (CGN) are transferred from 25 mM KCl (K25) to 5 mM KCl (K5) caspase-3 and caspase-8, but not caspase-1 or caspase-9,activities are induced and cells die apoptotically. CGN death was triggered by a [Ca(2+)](i) modification when [Ca(2+)](i) was reduced from 300

Activation of the peroxisome proliferato
โœ Steven A. Smith; Fiona J. May; Gregory R. Monteith; Sarah J. Roberts-Thomson ๐Ÿ“‚ Article ๐Ÿ“… 2001 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 105 KB ๐Ÿ‘ 1 views

## Abstract Peroxisome proliferatorโ€activated receptorโ€ฮฑ (PPARฮฑ) is a member of the steroid hormone receptor superfamily. In rodents, PPARฮฑ alters genes involved in cell cycle regulation in hepatocytes. Some of these genes are implicated in neuronal cell death. Therefore, in this study, we examined