๐”– Bobbio Scriptorium
โœฆ   LIBER   โœฆ

Depletion of nitric oxide synthase-containing neurons in the diabetic retina: reversal by aminoguanidine

โœ Scribed by E. Roufail; T. Soulis; E. Boel; M. E. Cooper; S. Rees


Publisher
Springer
Year
1998
Tongue
English
Weight
279 KB
Volume
41
Category
Article
ISSN
0012-186X

No coin nor oath required. For personal study only.


๐Ÿ“œ SIMILAR VOLUMES


Identification of alternate transcripts
โœ Thomas J. Giove; Monika M. Deshpande; William D. Eldred ๐Ÿ“‚ Article ๐Ÿ“… 2009 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 267 KB ๐Ÿ‘ 2 views

## Abstract Nitric oxide (NO) is a major signaling molecule in the retina and CNS, with physiological roles in every cell type in the retina. Previous work shows that neuronal nitric oxide synthase (nNOS) is an important source of NO in the vertebrate retina. There are distinct, active alternative

Cellular localization of neuronal nitric
โœ Neufeld, Arthur H.; Shareef, Shakeel; Pena, Janethe ๐Ÿ“‚ Article ๐Ÿ“… 2000 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 952 KB

The neuronal form of nitric oxide synthase (NOS-1) has been localized to several cell types in the retinas of experimental animals; however, localization in the human retina has not been definitive. By using in situ hybridization and immunohistochemistry, we have compared the cellular expression and

Induction of neuronal nitric oxide synth
โœ Masayuki Ikeda; Hiroshi Komachi; Ikuko Sato; Toshiyuki Himi; Tatsuhiko Yuasa; Se ๐Ÿ“‚ Article ๐Ÿ“… 1999 ๐Ÿ› John Wiley and Sons ๐ŸŒ English โš– 42 KB ๐Ÿ‘ 2 views

A free radical, nitric oxide (NO), besides being a messenger molecule in the brain, becomes a neurotoxin if overproduced. We recently reported that methylmercury (MeHg) induces neuronal NO synthase (nNOS) in Purkinje cells. In the present study, we examined the distribution and the mechanism of nNOS