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Deletion of the G2A receptor fails to attenuate experimental autoimmune encephalomyelitis

✍ Scribed by Inga Osmers; Sherry S. Smith; Brian W. Parks; Shaohua Yu; Roshni Srivastava; Jillian E. Wohler; Scott R. Barnum; Janusz H.S. Kabarowski


Book ID
113730412
Publisher
Elsevier Science
Year
2009
Tongue
English
Weight
534 KB
Volume
207
Category
Article
ISSN
0165-5728

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## Abstract During immune‐mediated demyelinating lesions, the endocannabinoid system is involved in the pathogenesis of both neuroinflammation and neurodegeneration through different mechanisms. Here we explored the cellular distribution of the CB1 receptor (CB1R) in the central nervous system (CNS